Tick-borne diseases can have many symptoms and attack many body systems at once. The symptoms often move around the body. Since many different kinds of organisms can be present in the blood and body fluids, it explains why there is a cluster of many symptoms. Several diseases can be present at the same time.
For a colorful review of symptoms, click here
For Dr. Burrascano's list of symptom of Lyme Disease and other Tick-borne diseases in a chart pdf format, click here, and turn to page 9-10.
THE FOLLOWING INFORMATION BREAKS DOWN SYMPTOMS SPECIFIC TO DIFFERENT ORGANISMS ASSOCIATED WITH INFECTION INJECTED INTO THE BLOOD STREAM BY A TICK. ALSO AlSO LISTED ARE OPPORTUNISTIC INFECTIONS THAT ARE COMMON IN PEOPLE WITH TICK-BORNE INFECTION DUE TO A SEVERELY COMPROMISED IMMUNE SYSTEM. Click Here for a general list of infectious diseases.
XMRV is now thought to be a result of laboratory contamination from mouse cells or DNA. XMRV (Xenotropic Murine Leukemia Virus-Related Virus) is a retrovirus. The virus can also be carried in the mouse's blood, and as ticks suck in a blood meal, the virus is ingested from the mouse's blood and then vomited into a human being that has a tick attachment that has fed on a mouse.
The Eight Types of Human Herpes Viruses are listed below: for more info Click Here
Human simplex virus type 1 (HSV1)
Human simplex virus type 2 (HSV2)
Varicella-zoster virus (HHV-3)
Epstein-Barr virus (HHV-4)
Human herpes virus 6 and 7 (HHV-6 and HHV-7)
Human herpes Virus 8 (HHV-8)
The condition has been associated with a spirochete that has been isolated from skin lesions of patients and from ticks. Based on DNA analysis, it is not the same species as the spirochete that causes Lyme disease. Relatively little is known about STARI, which is apparent by the qualified details below.Vectors of Borrelia lonestari is the Amblyomma americanum, the Lone Star tick. This is frequently found in Missouri and Kansas, and was identified by Dr. Masters in Missouri.
(CSD), also known as catscratch fever or subacute regional lymphadenitis, is a bacterial infection affecting lymph nodes that drain the sites of inoculation. Bartonella henselae, a gram-negative rod, is considered the principal etiologic agent.
Tick-borne rickettsioses are caused by obligate intracellular bacteria belonging to the spotted fever group (SFG) of the genus Rickettsia within the family Rickettsiaceae in the order Rickettsiales (276). These zoonoses are among the oldest known vector-borne diseases.
In fact, many of the rickettsiae we now identify as human pathogens were first identified in ticks several years or even decades before a conclusive association with human disease was demonstrated, including 7 of the 11 species or subspecies of tick-borne SFG rickettsiae confirmed as pathogens since 1984. Tick-borne rickettsial diseases, including Flinders Island spotted fever, Japanese spotted fever, and Astrakhan fever. These include infections caused by R. sibirica subsp. mongolitimonae, R. slovaca, R. aeschlimannii, and R. heilongjiangensis. Increasing reports of imported African tick bite fever among travelers. Other infections include: tick-borne rickettsial diseases, including Flinders Island spotted fever, Japanese spotted fever, and Astrakhan fever, Rickettsia rickettsii (Rocky Mountain spotted fever)
Myriad applied and basic research questions remain in the study of spotted fever rickettsiae and the diseases that these agents cause. A sampling of unresolved issues include the need for reliable, early diagnostic tests; development of serological assays that discriminate among various SFG rickettsial infections and provide an agent-specific diagnosis; better characterization of the natural histories of newly recognized SFG rickettsial pathogens; elucidating the pathogenic mechanisms (including rickettsial reactivation in the tick, the role and effect of tick saliva on the early infection, and the identification of rickettsial virulence genes); and prospective active surveillance studies that better the magnitude and distribution of various spotted fever rickettsioses.
The molecular adaptations required by pathogenic spirochetes for efficient transmission by obligate, blood-feeding ticks are largely unknown. In the new era of genomics, the complete DNA sequence of two spirochetes, Borrelia burgdorferi and Treponema pallidum, have been determined (1,2). As additional genome sequences become available for other pathogenic and free-living spirochetes, comparisons of their genomes may elucidate genes that are unique to those species of spirochetes associated with ticks. This information, along with an increased understanding of the molecular mechanisms used by tick-borne spirochetes to adapt for transmission by their tick vectors, may lead to unique disease prevention strategies.
The genus Borrelia currently contains 37 species of spirochetes, many of which cause diseases in humans and domestic animals (Table) (3). Except for Borrelia recurrentis (which causes louse-borne relapsing fever and is transmitted by the human body louse), all known species are transmitted by ticks (4). Two groups of spirochetes stand out among these tick-borne species because of their prevalence as human pathogens: Lyme disease spirochetes, transmitted by the relatively slow-feeding ixodid (hard) ticks of the genus Ixodes, and relapsing fever spirochetes, transmitted by the fast-feeding argasid (soft) ticks of the genus Ornithodoros (Figure 1). Major observations in recent years have increased our understanding of how one species in each group adapts while infecting ticks. B. burgdorferi, a causative agent of Lyme disease, and B. hermsii, a causative agent of tick-borne relapsing fever, have received the most attention. We describe how these two species of Borrelia change their outer surface during their alternating infections in ticks and mammals, which in turn suggests biological functions for a few surface-exposed lipoproteins. The dynamics of infection of these two bacteria in strikingly different types of ticks provide examples of possible adaptations for their transmission.
Click here for more information about Protozoa
Toxoplasma gondii is a species of parasitic protozoa in the genus Toxoplasma. The definitive host of T. gondii is the cat, but the parasite can be carried by many warm-blooded animals (birds or mammals, including humans). Toxoplasmosis, the disease of which T. gondii is the causative agent.
Symptoms of the infection vary.
If you have a weakened immune system, ask your doctor about having your blood tested for Toxoplasma.
It is an intracellular parasite that undergoes development within white blood cells, especially granulocytes.
Babesia is a protozoan parasite of the blood that causes a hemolytic disease known as Babesiosis. There are over 100 species of Babesia identified; however only a handful have been documented as pathogenic in humans.
Babesia is a protozoan parasite of which Babesia microti and Babesia divergens are the two species to most frequently infect humans. Infections from other species of Babesia have been documented in humans but are not habitually seen. Babesiosis is also known as Piroplasmosis. Due to historical misclassifications, this protozoan was labeled with many names that are no longer used. Common names of the disease include Texas Cattle Fever, Redwater Fever, Tick Fever, and Nantucket Fever.[
Symptoms of Babesiosis
An incubation period of one week to eight weeks elapses between the tick bite and the onset of Babesia symptoms. Almost everyone who contracts this disease gets flu-like symptoms of fever and chills.
Nonspecific symptoms include:
Specific symptoms of babesiosis (as consequences of ruptured red blood cells) include:
Having an infection that destroys red blood cells does a few particularly damaging things to the body:
2009-2010: Dr Eva Sapi, from the University of New Haven, Connecticut is doing research on ticks. So far, it appears that she has found worms in 40% of the ticks she has studied.
There is a antibody blood serum test available for Strongyloides stercoralis.
This article is an excerpt from the 2008 Lyme Disease Annual Report.
"Mold toxins are found in foods such as grains and peanuts. Far more serious are the mold toxins found in buildings with water leaks. Buildings with flat roofs and buildings on a concrete slab at the bottom of a hill are ready-made for mold problems. Mold toxins are the most common of the biotoxins and are responsible for many of the symptoms of Lyme Disease patients.
When a building has a water leak or water damage that is not immediately corrected, molds such as Stachybotrys, Aspergillus, Penicillium and Chae-tomium are likely to start colonies. The colonies may be visible or they may be hidden in places such as the tops of ceiling tiles or the bottoms of carpets. They send out spores which contain potent nerve toxins or neurotoxins.
When these spores with their neurotoxins are inhaled, about 76% of the population can make antibodies to the toxins and quickly eliminate them. They may sneeze, have a sore throat or have other minor symptoms, but symptoms are temporary. If they spend several days in a moldy building, they will begin to feel sick, but when they are away for a few days they recover. These same people are also able to eliminate Lyme toxins quickly.
About 24% of the population have a genetic makeup that makes them unable to produce the needed antibodies. The body simply does not identify the toxins as invaders and it does not eliminate them effectively. The liver can send them to the digestive tract via the bile, but they are quickly reabsorbed back into the blood. The result is that continual or repeated exposures to mold toxins result in an ever increasing amount of these toxins in the body. And if the person has Lyme Disease, Lyme toxins also build up in the body the same way. These are the people that get very sick when they do frequency treatments for Lyme. Frequency treatments for Lyme are not recommended until the toxin issue is addressed."
There is much more information available, more symptoms to discuss, but basically, people with tick borne diseases must live in a mold-free environment in order to get well. Mold toxins also cause increasing weight gain.
"The Mystery of Morgellon's Disease, Infection or Delusion?"
"Morgellons disease is a mysterious skin disorder that was first described more than 300 years ago. The disease is characterized by fiber-like strands extruding from the skin in conjunction with various dermatologic and neuropsychiatric symptoms. In this respect, Morgellons disease resembles and may be confused with delusional parasitosis. the association with Lyme disease and the apparent response to antibacterial therapy suggest that Morgellons disease may be linked to an undefined infectious process. Further clinical and molecular research is needed to unlock the mystery of Morgellons disease." by Virginia R. Savely, Mary M. Leitao, and Raphael B. Stricker. Click here to see the full article.